Smoking And Heart/atheriosclerosis
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SMOKING AND HEART/ATHEROSCLEROSIS
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Diet And Health: Common Sense For The Heart - "Still, the relationship between high fruit and vegetable intake and low risk of heart disease remained regardless of exercise or smoking habits and vitamin use." -- In yet another study that would never have been funded in a rational society, the benefits of a healthy diet are revealed. Fruits, vegetables and a diet low in fat are good for the heart. This is what Mother had in mind when she made you eat your spinach and gave you an apple instead of chocolate cake. Only in America would this study make news. Much is made of the so-called Mediterranean diet of nuts, grains, vegetables and fruits but low in meat. The Greeks, whose smoking are among the highest on the planet, exemplify the salutary effects of the Mediterranean diet. Studies conducted in rural areas in Greece indicate that negative effects from smoking are virtually non-existent.
BacterialInflammatory Protein Linked With Heart Disease - Italian researchers have foundanother link between inflammation and heart disease, they report intoday’s rapid access issue of Circulation: Journal of the American HeartAssociation. A protein that promotes inflammation and immune responsesknown as Chlamydia pneumonia heat shock protein 60 (Cp-HSP60) appears to bea strong indicator of heart disease and may lead to new ways to identifypeople at risk for it, says lead author Luigi Marzio Biasucci, M.D.,assistant professor of cardiology, Catholic University of Rome. “Ninety-nine percent of patients withacute coronary syndrome tested positive for Cp-HSP60 in their blood,” hesays.
|UPDATES LINKING EVIDENCE AND EXPERIENCE - Chlamydia pneumoniae and Atherosclerosis - Some "research" was done last year (1998) about intimal medial thickening and smoking and atherosclerosis. This article raises interesting questions, points out the limitations of its own findings and also has a lot of references that might bear investigation. If the link does not work, click here.|
|SMOKING DOES NOT CAUSE MYOCARDIAL INFARCTION - "Glucose level and systolic blood pressure were associated with the incidence of MI, but smoking and lipid measures were not." The American Medical Association seems to be conveniently quiet on this study, though published in its own archives. It does not take great intelligence to understand the reasons.|
|MONICA did not deliver on task it set out to accomplish - The title of this letter about the MONICA study (set out by the WHO to "prove" that smoking causes hearth disease) by Jeremy Laurance, Health editor of The Independent, London, and published by the BMJ, says it all.|
|OOPS! ANOTHER 'PUZZLING PARADOX' ABOUT SMOKING... - After the WHO's MONICA study, here is: TRENDS IN THE INCIDENCE OF MYOCARDIAL INFARCTION AND IN MORTALITY DUE TO CORONARY HEART DISEASE, 1987 TO 1994 - In this study it is very interesting to see that notwithstanding the sharp decrease in smoking, the rate of hospitalization for Coronary Heart Disease has actually increased. It is also interesting to see how the explanations for this "mystery" go in every possible direction, but the most instinctive and logical one: smoking does not cause CHD. But this would mean proving once again that the dogma dictating that tobacco is an absolute health evil is false, and this is, of course, not politically acceptable, for the grant money and the support of the pharmaceutical industry would be seriously compromised.|
|WORLD HEALTH ORGANIZATION: THE LARGEST CARDIOLOGICAL STUDY EVER PERFORMED FAILS TO FIND A LINK BETWEEN HEART ATTACK AND SMOKING - "The largest ever cardiology study has failed to find a link between heart attacks and the classic risk factors, such as smoking and high cholesterol levels." "The Monica study, which assessed 21 countries over 10 years, found the incidence of heart disease dropping across Europe, Australia and North America. But scientists could find no statistical connection between the reduction and changes in obesity, smoking, blood pressure or cholesterol levels."
THE WHO TRIES TO EXPLAIN THEIR UNEXPECTED RESULTS - Why does this massive study disprove anti-smoking dogmas" This may have political consequences, and the truth may even come out! A flabbergasted WHO tries to explain: "There are several possible explanations for our findings, including problems of measurement, the fact that rates were declining in most populations anyway, and lack of linearity in trends associated with possible time-lags for which preliminary analyses do not allow. Another interesting possibility is that in population terms the contribution of the classical risk factors is swamped by that of other, dietary, behavioural, environmental or developmental factors, of which several have been proposed since the study was launched." -- Above and beyond word spinning, the fact is just one: THERE IS NO ASSOCIATION BETWEEN SMOKING AND HEART ATTACKS. Period.
|The Risk of Coronary Heart Disease in non-smokers exposed to Environmental Tobacco Smoke - Research article - Dr. Alan J. Gross pulls no punches in this research article on ETS: "The toxicological evidence provided by Glantz and Parmley has been challenged by Wu and by Gori, among others. Moreover, the epidemiologic data considered by Glantz and Parmley are equivocal at best and do not include data from the American Cancer Society's Cancer Prevention Studies (CPS-I) and (CPS-II )and the National Mortality Followback Survey which, when added to the original epidemiologic database considered by Glantz and Parmley, indicate no statistically significant association."|
|Heart Disease - Applause to Niemel et al amd Whincup et al for recognizing the potential for confusion from adjusting for so-called established risk factors without thinking. All too often this is not the case. Like automatons, too many researchers control their analyses of Helicobacter pylori and Chlamydia pneumoniae infection (HP and CP) for social class and smoking, heedless of the fact that HP & CP may well account for those social class and smoking differences, and thus they should not be controlled for because this obliterates the truly key facts! This is the iron tyranny that has stifled scientific thought for 30+ years. Fortunately the tide has begun to turn. Unfortunately bacterial causes of CHD aren't very exciting "news," so we rarely read of them in the popular press.|
|ETS Heart Disease Study Is Epidemiologic Malpractice - This study is fraudulent, because the authors knowingly left out the risk factors that really cause excess heart disease in smokers and in passive smokers. And Howard's boast to the media that "We threw the kitchen sink at the data, and still couldn't make it go away," (Knight-Ridder) is an outright lie. This study and the editorial also fail to mention Helicobacter pylori and Chlamydia pneumoniae infection, which many studies have linked to cardiovascular disease. Nor is there mention of C-reactive protein, a general marker of inflammation.|
|Chlamydia Pneumoniae: Risk Factors for Seropositivity and Association with Coronary Heart Disease - This is the last of a trinity of studies we publish on Helicobacter pylori and Chlamydia pneumoniae infections, responsible for much of the disease which is instead attributed to smoking by certain incompetent or corrupted medical organizations, and state-funded antismoking propaganda. "Serological evidence of C. pneumoniae infection is common amongst healthy British subjects. Smoking and social class are not important confounding variables in this study. Reinfection from contact with infected children in the home may be important in inducing higher titres in some subjects." - Link to MEDLINE.|
|Relation of Helicobacter Pylori Infection and Coronary Heart Disease - Smoking has been blamed for heart disease for the longest time, and indeed it is a contributor -- one of 300 known so far. How these contributors interact with each other in each individuals no serious scientist can tell, unless, of course, he is part of the antismoking racket in which case, the answer is always the same. This week, we link to MEDLINE for an abstract of this study on what is strongly emerging as an important contributor to heart disease, and which -- once again -- has nothing to do with smoking.|
|Association of Helictobacter Pylori and Chlamydia Preumoniae Infections with Coronary Heart Disease and Cardiovascular Risk Factors - Smoking has been blamed for heart disease for years, and indeed it is a contributor -- one of 300 known so far. How these contributors interact with each other in each individuals no serious scientist can tell, unless, of course, if part of the antismoking racket in which case, the answer is always the same. We link to the BMJ for this study on what more and more surfaces to be an important contributor to heart disease which has nothing to do with smoking.|
|Environmental Tobacco Smoke And Coronary Heart Syndromes: Absence Of An Association - The title says it all. This study from Prof. Gori, long associated with the tobacco industry, validly demonstrates its point.|
|Ischemic Heart Disease And Spousal Smoking In The National Mortality Followback Survey - "No overall spousal smoking-heart disease risk elevation was observed for either males or females", this study says.|
|Smoking Does Not Cause Congenital Heart Defects - Bibliography and results graph chart of 13 recent studies demonstrating the lack of relationship between smoking and congenital heart defects.
|Smoking and The Decline in Heart Attack Deaths - At a superficial glance, declines in smoking and declines in ischemic heart disease (heart attack or myocardial infarction) appear to correlate. From 1963 (the peak year) to 1985, the age-adjusted death rate fell by 42%. But the decline in IHD has been much greater than the decline in smoking. And when the data are analyzed by sex, the discrepancy is even more marked.|