Antis claim that smoking bans are absolutely necessary to preserve the health of hospitality workers because SG Carmona said in a press conference that "there is no safe level of exposure to SHS."
However, that is not exactly what his 2006 report said!
The report said:"evidence indicates that there is no risk-free level of exposure to secondhand smoke".
So,just what level of risk is there from exposure to SHS?
The level of risk runs from SLIM(20%) to NONE(almost non-existent)!!!!
The chances that a smoking ban will make a difference in preserving the health of hospitality workers are SLIM TO NONE!!!!!
The Health Consequences of Involuntary Exposure to Tobacco Smoke
Page 10, Chapter 1
Specificity, referring to a unique exposure-disease relationship (e.g., the association between thalidomide use during pregnancy and unusual birth defects), can be set aside as not relevant, as all of the health effects considered in this report have causes other than involuntary smoking.
Introduction, Summary, and Conclusions, page 11
Major Conclusions
4. The scientific evidence indicates that there is no risk-free level of exposure to secondhand smoke.
Page 14, Chapter 1
Respiratory Symptoms and Prevalent Asthma
in School-Age Children
7. The evidence is sufficient to infer a causal relationship between parental smoking and ever having asthma among children of school age.
( NOTE: The RR for this is 1.23, that means there is an 80% chance it was not caused by parental smoking.)
Childhood Asthma Onset
9. The evidence is suggestive but not sufficient to infer a causal relationship between secondhand smoke exposure from parental smoking and the onset of childhood asthma.
(NOTE:Therefore,the level of risk is almost none.)
Introduction, Summary, and Conclusions Pages 15-16
Lung Cancer
2. The pooled evidence indicates a 20 to 30 percent increase in the risk of lung cancer from secondhand smoke exposure associated with living with a smoker.
(NOTE: RR1.25 shows that there is an 80% chance a non-smokers lung cancer was caused by risk factors other than SHS exposure.)
Chapter 8. Cardiovascular Diseases from Exposure to Secondhand Smoke
2. Pooled relative risks from meta-analyses indicate a 25 to 30 percent increase in the risk of coronary heart disease from exposure to secondhand smoke.
(NOTE: RR1.25 shows that there is an 80% chance a non-smokers heart disease was caused by risk factors other than SHS exposure.)
3. The evidence is suggestive but not sufficient to infer a causal relationship between exposure to secondhand smoke and an increased risk of stroke.
(NOTE: Risk level is almost non-existent.)
4. Studies of secondhand smoke and subclinical vascular disease, particularly carotid arterial wall thickening, are suggestive but not sufficient to infer a causal relationship between exposure to secondhand smoke and atherosclerosis(hardening of the arteries).
(NOTE: Risk level is almost non-existent.)
Chapter 9. Respiratory Effects in Adults from Exposure to Secondhand Smoke
3. The evidence is suggestive but not sufficient to conclude that persons with nasal allergies or a history of respiratory illnesses are more susceptible to developing nasal irritation from secondhand smoke exposure.
(NOTE: Risk level is almost non-existent.)
Respiratory Symptoms
4. The evidence is suggestive but not sufficient to infer a causal relationship between secondhand smoke exposure and acute respiratory symptoms including cough, wheeze, chest tightness, and difficulty breathing among persons with asthma.
(NOTE: Risk level is almost non-existent.)
5. The evidence is suggestive but not sufficient to infer a causal relationship between secondhand smoke exposure and acute respiratory symptoms including cough, wheeze, chest tightness, and difficulty breathing among healthy persons.
(NOTE: Risk level is almost non-existent.)
6. The evidence is suggestive but not sufficient to infer a causal relationship between secondhand smoke exposure and chronic respiratory symptoms.
(NOTE: Risk level is almost non-existent.)
Lung Function
7. The evidence is suggestive but not sufficient to infer a causal relationship between short-term secondhand smoke exposure and an acute decline in lung function in persons with asthma.
(NOTE: Risk level is almost non-existent.)
8. The evidence is inadequate to infer the presence or absence of a causal relationship between short-term secondhand smoke exposure and an acute decline in lung function in healthy persons.
(NOTE: Risk level is almost non-existent.)
9. The evidence is suggestive but not sufficient to infer a causal relationship between chronic second-hand smoke exposure and a small decrement in lung function in the general population.
(NOTE: Risk level is almost non-existent.)
10. The evidence is inadequate to infer the presence or absence of a causal relationship between chronic secondhand smoke exposure and an accelerated decline in lung function.
(NOTE: Risk level is almost non-existent.)
Asthma
11. The evidence is suggestive but not sufficient to infer a causal relationship between secondhand smoke exposure and adult-onset asthma(asthma attack).
(NOTE: Risk level is almost non-existent.)
12. The evidence is suggestive but not sufficient to infer a causal relationship between secondhand smoke exposure and a worsening of asthma control.
(NOTE: Risk level is almost non-existent.)
Chronic Obstructive Pulmonary Disease(Emphysema and other stuff)
13. The evidence is suggestive but not sufficient to infer a causal relationship between secondhand smoke exposure and risk for chronic obstructive pulmonary disease.
(NOTE: Risk level is almost non-existent.)
14. The evidence is inadequate to infer the presence or absence of a causal relationship between secondhand smoke exposure and morbidity(symptoms) in persons with chronic obstructive pulmonary disease.
(NOTE: Risk level is almost non-existent.)